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Whether you live in an apartment or a house or just want to keep your home free from EMFs, there are a number options to limit your exposure. One of the easiest is to limit the usage of your electronic devices. It is also possible to use EMF block emf paint to stop EMF radiation from entering your house. Another easy way to protect your house against EMF radiation is to install an RF shielding canopy. It’s a kind made of net which contains EMF shielding. It’s used to stop EMFs from entering a space. Another option is to get your home equipped with a conductive enclosure. These devices are called Faraday cages.
Several studies have shown studies have shown that the nonionizing EMF has anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF’s anticancer activity in vitro is thought to involve down-regulation of cancer stem cells. This could be the reason for the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism of AM EMF’s impact on patients with cancer is not yet clear.
The effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were examined in mice. The tumors were divided into three groups. One group did not have exposure to RF EMF. The second group was exposed RF EMF at the same frequency to the frequency used by humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumors was assessed against the effect of RCF. The results indicated that tumors treated by HCCMF showed significant shrinkage. However, the tumours treated with RCF showed no evidence of tumour shrinkage.
The reason for tumor-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF’s antiproliferative effect on HCC cells is controlled by CACNA1H, a protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have more broader implications for the diagnosis and treatment of various cancers.
The tumours in the controls were never exposed EMF from RF, and fed a standard mouse diet. The tumors in HCCMF HCCMF group were injected with Huh7 cells at the time they were five-seven weeks old. The tumors were removed in cases of excessive burden.
The tumors in the three groups also displayed different growth curves. The HCCMF-treated tumors showed a significant decrease in the size of the tumour after eight weeks. However, the tumors treated with RCF didn’t show shrinkage. The difference was significant. The tumors treated by RCF showed necrosis that is common when tumors are exposed to RCF. It is possible that this necrosis was due to a lack of oxygen in the more invasive tumours.
In sum, the results indicate that AM RF EMF has anticancer activity in vitro and in the vivo. Numerous studies have demonstrated that AM RF EMF produces measurable reduction in tumours within HCC patients. The possibility is that the AM EMF triggers these effects through CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in the vivo.