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Whether you live in a house or apartment, or you just need to ensure that your house is free of electromagnetic fields There are a variety of methods to reduce exposure. One of the easiest is to limit your electronic device use. You could also consider EMF block paint to prevent EMF radiation from reaching your house. Another easy way to protect your house against EMF radiation would be to use an RF shielding canopy. It is a type of net that contains EMF shielding. It’s used to stop EMFs from entering a room. Another alternative is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism behind AM RF EMF’s anticancer activity in vitro is thought to result from the deregulation in cancer-related stem cells. This could explain the long-term responses seen in certain patients suffering from advanced HCC. However, the reason behind AM RF EMF’s effect in cancer patients is not clear.
The effects from AM RF EMF on HCC tumour growth in vivo were studied in mice. The tumors were classified into three groups. The first group was not exposed RF EMF. The second group was exposed RF EMF at a frequency that is similar to that of humans. The third group was exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumors was assessed against the effect of RCF. The results showed that the cancers treated with HCCMF showed significant shrinkage. However, tumours treated with RCF did not show any evidence of shrinkage of the tumor.
The reason for tumour-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF’s antiproliferative effects on HCC cells is caused by CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have more broader implications in the diagnosis and treatment of various cancers.
The tumors in the controls were never exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumors of those in the HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were then killed after they had a high burden.
The tumors in the three groups also showed different growth curves. The HCCMF-treated tumors saw a significant decrease in size of the tumor after eight weeks. However, the tumours that were treated using RCF did not show any shrinkage. The difference was highly significant. The tumors treated by RCF were able to show necrosis, which is typical in tumours exposed to RCF. There is a possibility that the necrosis was due to an absence of oxygen in larger cancers.
In summary, the results show the fact that AM EMF is a powerful source of anti-cancer properties in vitro as well as in vivo. Several studies have shown that AM RF emf blockers produces measurable tumour shrinkage in HCC patients. There is a possibility that AM RF EMF produces these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained impact on the growth of HCC tumours in the vivo.