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Whether you live in an apartment or house or simply would like to keep your home free of electromagnetic fields There are plenty options to limit your exposure. One of the most straightforward is to limit the usage of your electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your home. Another method to protect your house from EMF radiation would be to put up a RF shielding canopy. It is a type of net that contains EMF shielding and is used to stop EMFs from entering a room. Another option is to have your home equipped with an electrical enclosure. These enclosures are known as Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF can cause antiproliferative effects in HCC cells. The mechanism of AM RF EMF’s anticancer activity in vitro is thought to result from the deregulation the cancer stem cell. This could explain the long-term effects observed in patients with advanced HCC. However, the mechanism of AM EMF’s impact on patients with cancer is not yet clear.
The effects on the effects of AM RF EMF on HCC tumor growth in vivo were examined in mice. The tumors were separated into three groups. One group did not have exposure to RF EMF. Another group of participants was subjected to RF EMF at a frequency that is similar to the frequency used by humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumors was compared to that of RCF. The results showed that the tumours treated with HCCMF showed significant shrinkage. However, the tumors treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism behind tumour-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF’s ability to inhibit proliferation on HCC cells is caused by CACNA1H, a protein that is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications in the diagnosis and treatment of various cancers.
The tumours in those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumors of the HCCMF group were injected with Huh7 cells after they were between five and seven weeks old. The tumours were then euthanized in cases of excessive burden.
The tumors of the three groups also displayed different growth curves. The tumors treated with HCCMF had a significant reduction in size of the tumor after eight weeks. However, tumors that were treated using RCF did not show any shrinkage. The difference was highly significant. The tumors treated with RCF showed necrosis, which is common in tumors that have been that are exposed to RCF. There is a possibility that the necrosis was caused by the lack of oxygen in the more invasive tumors.
In sum, the results show the fact that AM EMF is a powerful source of anticancer effects in vitro and in live. A number of studies have proven it is true that AM RF EMF produces measurable tumour shrinkage for HCC patients. It is possible that AM RF EMF triggers these effects due to CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in the vivo.