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If you reside in an apartment or a house or simply would like to keep your home free from EMFs There are a variety options to limit your exposure. One of the easiest is to reduce the use of electronic devices. You can also turn to EMF block emf paint to prevent EMF radiation from entering your home. Another way to shield your home from EMF radiations is to put up an RF shielding cover. This is a cloth of net that contains EMF shielding and is used to prevent EMFs from entering a room. Another option is to get your house equipped with a conductive enclosure. These enclosures are known as Faraday cages.
Numerous studies have demonstrated studies have shown that the nonionizing EMF can cause antiproliferative properties in HCC cells. The mechanism of AM RF EMF’s anticancer activity in vitro is believed result from the deregulation the cancer stem cell. This could explain the long-term effects observed in patients with advanced HCC. But, the reason for AM EMF’s impact on cancer patients is not clear.
Effects on the effects of AM RF EMFs on HCC tumor growth in vivo were studied in mice. The tumours were divided into three groups. One group did not have exposure to RF EMF. Second group members were exposed to RF EMF at the same frequency to the frequency used by humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was evaluated against that of RCF. The results showed that the tumors treated by HCCMF were significantly shrinking. However, the tumours treated with RCF showed no evidence of tumour shrinkage.
The mechanism behind cancer-specific AM RF EMF could be driven by the fact that tumour cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF’s antiproliferative effects in HCC cells is caused through CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications in the diagnosis and treatment of a variety of cancers.
The tumours in the control group were not exposed EMF from RF, and fed a standard mouse diet. The tumors of HCCMF HCCMF group were injected with Huh7 cells when they were between five and seven weeks old. The tumors were then killed when they showed excessive burden.
The tumors in the three groups showed different growth curves. The HCCMF-treated tumors saw a significant decrease in the size of the tumour after 8 weeks. However, tumors which were treated by RCF didn’t show reduction in size. The difference was highly significant. The tumors treated by RCF showed necrosis, which is typical in tumors that have been exposed to RCF. It is possible that the necrosis was due to an absence of oxygen in the more invasive tumors.
In sum, the results suggest an AM-RF EMF exhibits anticancer effects in vitro and in vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage in HCC patients. There is a possibility that AM RF EMF produces these effects due to CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting impact on the growth of HCC tumors in living tissue.