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Whether you live in a house or apartment or just would like to keep your home free from EMFs There are plenty options to limit your exposure. One of the most effective is to limit the usage of your electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from reaching your home. Another easy way to protect your house from EMF radiation is to use an RF shielding cover. It is a type made of net which contains EMF shielding. It’s used to block EMFs from entering rooms. Another option is to get your home fitted with a conductive enclosure. These devices are called Faraday cages.
A number of studies have proven studies have shown that the nonionizing EMF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF’s anticancer activity in vitro is believed be based on the downregulation the cancer stem cell. This could be the reason for the long-term effects observed in patients with advanced HCC. However, the reason behind AM RF EMF’s effect in cancer patients isn’t evident.
Effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumours were divided in three different groups. First, the group that was unaffected to RF EMF. Second group members were exposed to RF EMF at the same frequency to that used in humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumors was evaluated against that of RCF. The results indicated that tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism of tumor-specific AM RF EMF could be driven by the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF’s antiproliferative effect in HCC cells is mediated by CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have more broader implications in the treatment and diagnosis of various cancers.
The tumors in the control group were not exposed EMF from RF, and fed a normal mouse diet. The tumors in HCCMF HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. The tumours were then euthanized in cases of excessive burden.
The tumors in the three groups also displayed different growth curves. The tumors treated with HCCMF saw a significant decrease in the size of the tumour after 8 weeks. However, tumors which were treated by RCF did not show any reduction in size. The difference was significant. The tumors treated by RCF had necrosis, which is typical when tumors are that are exposed to RCF. There is a possibility that this necrosis is caused by the lack of oxygen in larger cancers.
In conclusion, the findings show the fact that AM EMF exhibits anti-cancer properties in vitro as well as in live. Numerous studies have demonstrated the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. The possibility is that the AM EMF triggers these effects because of CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF emf blockers may cause a lasting impact on the development of HCC tumours in vivo.