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No matter if you live in an apartment or house or just want to keep your home free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the easiest is to restrict the usage of your electronic devices. It is also possible to use EMF block paint to stop EMF radiation from entering your home. Another way to shield your home against EMF radiation is to install a RF shielding canopy. It is a type made of net which contains EMF shielding. It’s used to block EMFs from entering a space. Another option is to get your home fitted with a conductive enclosure. These enclosures are known as Faraday cages.
A number of studies have proven how the EMF that is not ionized can cause antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF’s anticancer activity in vitro is thought to involve down-regulation the cancer stem cell. This may account for the long-term effects observed in some patients with advanced HCC. But, the reason for AM RF EMF’s effect in cancer patients is not clear.
Effects on the effects of AM RF EMF on HCC tumour growth in vivo was studied in mice. The tumors were divided into 3 groups. First, the group that was unaffected RF EMF. Another group of participants was subjected RF EMF at the same frequency to that used in humans. The third group was exposed the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was compared to that of RCF. The results revealed that tumors treated by HCCMF were significantly shrinking. However, the tumours treated with RCF didn’t show evidence of shrinkage of the tumor.
The mechanism of tumor-specific AM RF EMF may be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF’s antiproliferative effect on HCC cells is caused through CACNA1H which is a protein which mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications for the diagnosis and treatment of various cancers.
The tumours of those in the group that were unaffected to RF emf blockers, and were fed a normal mouse diet. The tumours in those in the HCCMF group were infected with Huh7 cells after they were five-seven weeks old. The tumors were then killed in cases of excessive burden.
The tumors in the three groups also displayed different growth curves. The tumours treated with HCCMF showed a significant decrease in size of the tumor after 8 weeks. However, the tumors which were treated by RCF did not show any shrinkage. The difference was significant. The tumors treated by RCF showed necrosis, which is common in tumors that have been exposed to RCF. The possibility is that this necrosis is caused by a lack of oxygen in the larger cancers.
In summary, the results indicate that AM RF EMF exhibits anti-cancer properties in vitro as well as in the vivo. Several studies have shown the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. It is possible that AM RF EMF produces these effects because of CACNA1H, a protein that is involved in the tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained impact on the development of HCC tumours in the vivo.